04/12/2021 3:55 PM
Updated 04/12/2021 16:10
It would be easier to explain the death of journalist and driver Mauro Viale if there were not a tangle of questions surrounding the coronavirus.
The most elementary question will be answered soon, when the medical part of the Los Arcos clinic explains how was the chain of events that in the blink of an eye transformed
into cardiorespiratory arrest
The other questions are more complex: How common are the adverse effects of Covid on the cardiovascular system?
Who do they get?
The experts of the involved specialties (virology, infectology, cardiology) know of the close relationship between viral infections and cardiovascular diseases.
It's not new.
Myocarditis was seen as a consequence of SARS1 and it is already assumed that the cross between SARS-CoV-2 and, for example,
cerebrovascular accidents (CVA) are nine times more frequent than those that occur due to influenza, that is, flu.
In fact, the Argentine Cardiological Foundation reports that 2% of patients who arrived at hospitals with Covid had stroke.
In half of those cases, stroke was their first coronavirus symptom.
45% had no associated risk factor.
The photo that Alberto Fernández uploaded to his Instagram to fire Mauro Viale
But what happened to Viale?
Rumors are running, in the absence of an official report.
At this time, the presence of
that would have caused Viale's death
is hinted at
A relatively common picture of pulmonary embolism (or
You have to go back a year to read the first reports that spoke of "
" in the lungs of people who died from Covid.
Once generated, the clot travels through the circulatory system and is capable of obstructing regular flow at the brain or heart level.
In fact, the proliferation of these cases led to one of the almost “mandatory” medications of patients hospitalized for Covid being
Did Viale receive that medication or what happened to him?
A source linked to the case, who asked to reserve his name, confirmed that the journalist “had been vaccinated on Thursday and on Friday night he had a fever.
On Saturday he was admitted for desaturation with bilateral pneumonia, which was corrected with low-flow oxygen.
After being monitored in intensive care until noon on Sunday,
with total stability
and adequately saturating with very little oxygen, he went to the floor ”.
Microscopic image of the coronavirus, courtesy of the National Institute of Allergy and Infectious Diseases in the United States.
Then, "without fever, saturating well and interacting with doctors, physiotherapists and talking on the phone, which he did permanently from the moment he was admitted to the sanatorium, he suffered a picture of
that could not be reversed despite the resuscitation maneuvers."
Leaving aside the absurd questions that were suggested in these hours regarding vaccination, the indications, as outlined above, point to a picture of
that, in one way or another, went unnoticed in front of medical personnel, and that is already frequent in cases of Covid.
So much so that in April 2020, a study by the Spanish university hospitals Vall d'Hebron and Germans Trias i Pujol, described that
one in four serious patients with coronavirus had venous or pulmonary thrombosis
However, the condition was asymptomatic in 62% of the patients.
had deepened in August about the challenge that doctors around the world are representing to determine in time the effects and silent sequelae of Covid in cardiovascular matters, even in patients who go through the infection without symptoms.
biochemist, researcher at the Institute for Biomedical Research on Retroviruses and AIDS (INBIRS, from the UBA-CONICET), explained that “
the cases of myocarditis in patients with Covid are a concrete fact
In a recent review of clinical analyzes, it was reported that in 26 studies that included 11,685 patients, the prevalence of acute myocardial injury was
, although it ranged from 5% to 38% depending on the criteria used. "
"Now, the reasons that trigger it are not totally clear and different possibilities are being considered that are not mutually exclusive," he added.
At this point, Tartaglione clarified that "myocarditis and thrombosis are not the same thing ... while myocarditis cases can be caused by influenza or Covid, in Viale's case there seems to be something
, which generated a thrombosis."
The "why" of these pictures could not be fully answered by the experts.
The biggest doubt is
whether the thrombi are a "direct" consequence of Covid or, instead, a product of the so-called "cytokine storm"
, that kind of "self-attack" of the body, caused by an excessive response against SARS.
Quarleri explained that part of the debate is which is the cause and which is the consequence, but "the literature contemplates the possibility of 'linking' myocardial injury with hypercoagulability and thrombi".
As he clarified, coagulopathy in infected patients can be caused by disorders at the level of the cells that line our blood vessels (vascular endothelium), and that when infected with SARS-Cov-2 try to “repair themselves”, an event that could trigger a
cascade of coagulation
On the other hand, this "cascade" can be magnified by the body's own immune response.
The "targets" of these clots can be diverse.
Undoubtedly, it is worth asking (as Quarleri wondered) if it is possible that a non-critical patient like Mauro Viale has a pulmonary embolism.
The answer is certain: "Definitely yes, as I could have had a stroke or an acute myocardial infarction. These are consequences that can occur."
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